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Kaj doslej vemo o prehrani LCHF?

Moderatorji: UrosS, Metka, Stasa, Grom

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Grom

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Šefe

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Pridružen: Če sep 23, 2004 18:35

Odgovor To jan 10, 2017 14:12

Kaj doslej vemo o prehrani LCHF?

Kaj doslej vemo o prehrani LCHF?
Kdorkoli se je resno poukvarjal s prehrano LCHF, je zagotovo že slišal za profesorja Tima Noaksa. Prav v začetku leta je profesor skupaj s kanadskim kolegom Johannom Windtom objavil pregledni članek, ki je skorajda obvezno čtivo za vsakogar, ki ga zanima ta tema.
V članku, ki je v celoti dostopen v reviji British Journal of Sports Medicine (http://bjsm.bmj.com/content/51/2/133.full…), je lepo povzeto vse, kar je znanost do danes ugotovila o ko
ristih prehrane LCHF. V članku je opisano, kaj pomeni prehranjevanje po načelih LCHF, zakaj je taka prehrana učinkovita za hujšanje, kako lahko pomaga bolnikom s sladkorno boleznijo tipa 2 in ugodni vplivi prehrane LCHF na biomarkerje, s katerimi ocenjujemo dejavnike tveganja za nastanek srčno-žilnih bolezni. Popiše tudi ugoden vpliv prehrane na nealkoholno maščobno bolezen jeter v povezavi z aterogenimi dislipidemijami ter predstavi ključne zmote, ki jih imamo o prehrani LCHF.

V zaključku avtorja predlagata spremembo smernic klinične prehrane na podlagi spoznanj o prehrani z malo ogljikovimi hidrati in veliko maščobami.
Vir:
Noakes, T. & Windt, J., 2017. Evidence that supports the prescription of low-carbohydrate high-fat diets: a narrative review. Br J Sports Med, Izvod 51, pp. 133-139.

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Grom

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Šefe

Vnosov: 63851

Pridružen: Če sep 23, 2004 18:35

Odgovor To jan 10, 2017 14:34

Re: Kaj doslej vemo o prehrani LCHF?

Abstract

Low-carbohydrate high-fat (LCHF) diets are a highly contentious current topic in nutrition. This narrative review aims to provide clinicians with a broad overview of the effects of LCHF diets on body weight, glycaemic control and cardiovascular risk factors while addressing some common concerns and misconceptions. Blood total cholesterol and LDL-cholesterol concentrations show a variable, highly individual response to LCHF diets, and should be monitored in patients adhering to this diet. In contrast, available evidence from clinical and preclinical studies indicates that LCHF diets consistently improve all other markers of cardiovascular risk—lowering elevated blood glucose, insulin, triglyceride, ApoB and saturated fat (especially palmitoleic acid) concentrations, reducing small dense LDL particle numbers, glycated haemoglobin (HbA1c) levels, blood pressure and body weight while increasing low HDL-cholesterol concentrations and reversing non-alcoholic fatty liver disease (NAFLD). This particular combination of favourable modifications to all these risk factors is a benefit unique to LCHF diets. These effects are likely due in part to reduced hunger and decreased ad libitum calorie intake common to low-carbohydrate diets, allied to a reduction in hyperinsulinaemia, and reversal of NAFLD. Although LCHF diets may not be suitable for everyone, available evidence shows this eating plan to be a safe and efficacious dietary option to be considered. LCHF diets may also be particularly beneficial in patients with atherogenic dyslipidaemia, insulin resistance, and the frequently associated NAFLD.

Evidence that supports the prescription of low-carbohydrate high-fat diets: a narrative review
Abstract

Low-carbohydrate high-fat (LCHF) diets are a highly contentious current topic in nutrition. This narrative review aims to provide clinicians with a broad overview of the effects of LCHF diets on body weight, glycaemic control and cardiovascular risk factors while addressing some common concerns and misconceptions. Blood total cholesterol and LDL-cholesterol concentrations show a variable, highly individual response to LCHF diets, and should be monitored in patients adhering to this diet. In contrast, available evidence from clinical and preclinical studies indicates that LCHF diets consistently improve all other markers of cardiovascular risk—lowering elevated blood glucose, insulin, triglyceride, ApoB and saturated fat (especially palmitoleic acid) concentrations, reducing small dense LDL particle numbers, glycated haemoglobin (HbA1c) levels, blood pressure and body weight while increasing low HDL-cholesterol concentrations and reversing non-alcoholic fatty liver disease (NAFLD). This particular combination of favourable modifications to all these risk factors is a benefit unique to LCHF diets. These effects are likely due in part to reduced hunger and decreased ad libitum calorie intake common to low-carbohydrate diets, allied to a reduction in hyperinsulinaemia, and reversal of NAFLD. Although LCHF diets may not be suitable for everyone, available evidence shows this eating plan to be a safe and efficacious dietary option to be considered. LCHF diets may also be particularly beneficial in patients with atherogenic dyslipidaemia, insulin resistance, and the frequently associated NAFLD.
Vabljeni na moje spletne strani: www.gaspergrom.com

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