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Danes je Ne nov 17, 2019 03:59

Keto/Low carb študije

Moderatorji: UrosS, Metka, Stasa, Grom

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Grom

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Šefe

Vnosov: 63865

Pridružen: Če sep 23, 2004 18:35

Odgovor Pe jul 19, 2019 20:43

Re: Keto/Low carb študije

Limfa, limfne žleze, limfedem in keto / LCHF

Eno od novejših odkritij je razkrilo, da je za regulacijo in rast limfnih žil esencialnega pomena acetil koencim A, ena od spojin, ki nastaja med presnovo. Preprosto povedano, acetil koencim A lahko sproži rast limfnih žil in izboljša njihovo delovanje tudi v bolezenskih razmerah, kot je na primer limfedem. Ker acetil koencim A lahko nastaja iz ketonov
, se je znanstvenikom porodila ideja, da bi nemara dodajanje ketonov povečalo razpoložljivo količino acetil koencima A in ugodno vplivalo na limfatični sistem.

To zanimivo idejo so sprva preskusili na človeških celicah v epruveti, potem pa še na živih miškah, ki so jim predpisali ketogeno dieto – in pri obeh se je izkazalo, da deluje.




https://www.lchflove.si/

2019/07/15/

limfa-limfne-zleze-limfedem

-in-keto-lchf/






Slika
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Grom

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Šefe

Vnosov: 63865

Pridružen: Če sep 23, 2004 18:35

Odgovor To jul 30, 2019 09:41

Re: Keto/Low carb študije

12 tednov ketogene diete je ne samo stabilziralo diabetes tipa 2, temveč je stanje reverzibiralo. Glikiran hemoglobin se je po 26 letih po diagnozi diabetesa vrnil v normalne vrednosti! Da ne govorimo o pozitivnem pogledu na življenje in odpravi depresije!

In potem še vedno vidim članek, da to ni prava prehrana za diabetike :)

Ketogenic diets potentially reverse Type II diabetes and ameliorate clinical depression: A case study.
Cox N1, Gibas S1, Salisbury M1, Gomer J2, Gibas K3.
Author information
Abstract

Efficacious adherence to treatment protocol predicts metabolic control among Type 2 diabetics (T2DM) [1-4]; however, few healthcare systems employ individualized strategies to mediate the comorbidity of T2DM with other chronic disease states. A clinically prescribed ketogenic diet, patient-centered nutritional education and high intensity interval training (HIIT), girded by solution-focused psychotherapy, modulate significant improvements in the clinical biomarkers associated with concurring T2DM and clinical depression [5-15]. Relevant metabolic change was noted in the following measures: HOMA-IR, triglyceride/HDL ratio, HgA1c, fasting insulin, fasting glucose, fasting triglycerides, LDL, VLDL, HDL, total cholesterol and C-reactive protein. The Patient Health Questionnaire 9 (PHQ-9) along with clinical interview and the mental status exam showed notable change in the patient's depressive symptoms; likewise, her self-efficacy score normalized, as measured by the General Self-Efficacy Questionnaire (GSE) and the Metabolic Syndrome Compliance Questionnaire (MSC). The case study highlights a 65-year old female who presented with a 26-year history of dually-diagnosed Type 2 diabetes (T2DM) and major depressive disorder (MDD). The patient was prescribed a ketogenic diet (KD), clinically formulated from her resting metabolic rate, body fat percentage and lean body mass, together with weekly nutrition education, high intensity interval training (matched to her cardiovascular conditioning), and eight 45-minute solution-focused psychotherapy sessions. Intervention goals included improved insulin sensitivity evaluated by the HOMA-IR, sustained glycemic control measured via HgA1c, reduced cardiovascular risk via the triglyceride/HDL ratio, and improved depressive symptoms with increased self-efficacy monitored by the PHQ-9 and GSE/MSC. The results of the 12-week intervention were statistically significant. The patient's HgA1c dropped out of diabetic range (8.0%) and normalized at 5.4%. Her average daily glucose measurements declined from 216 mg/dL to 96 mg/dL; the HOMA-IR and triglyceride/HDL ratios improved by 75%. Her marker for clinical depression and measurement of self-efficacy normalized. The 12-week individualized treatment intervention served to functionally reverse 26 years of T2DM, ameliorate two and half decades of chronic depressive disorder and empower/equip the patient with a new experience of hope and success.
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Grom

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Šefe

Vnosov: 63865

Pridružen: Če sep 23, 2004 18:35

Odgovor Pe avg 02, 2019 08:12

Re: Keto/Low carb študije

Več raziskav kaže na pozitiven vpliv ketogene diete pri zmanjševanju pojava kroničnih glavobolov. To seveda vidimo tudi v praksi. Toda, kot kažejo moje izkušnje za optimum ni pomembna le LCHF prehrana temveč stanje visoke ketoze. Torej stroga ketogena dieta. Podobno v raziskavi:

Nutrients. 2019 Jul 28;11(8). pii: E1742. doi: 10.3390/nu11081742.
A Randomized Double-Blind, Cross-Over Trial of very Low-Calorie Diet in Overweight Migraine Patients: A Possible Role for Ketones?


Here we aimed at determining the therapeutic effect of a very low-calorie diet in overweight episodic migraine patients during a weight-loss intervention in which subjects alternated randomly between a very low-calorie ketogenic diet (VLCKD) and a very low-calorie non-ketogenic diet (VLCnKD) each for one month. In a nutritional program, 35 overweight obese migraine sufferers were allocated blindly to 1-month successive VLCKD or VLCnKD in random order (VLCKD-VLCnKD or VLCnKD-VLCD). The primary outcome measure was the reduction of migraine days each month compared to a 1-month pre-diet baseline. Secondary outcome measures were 50% responder rate for migraine days, reduction of monthly migraine attacks, abortive drug intake and body mass index (BMI) change. Only data from the intention-to-treat cohort (n = 35) will be presented. Patients who dropped out (n = 6) were considered as treatment failures. Regarding the primary outcome, during the VLCKD patients experienced -3.73 (95% CI: -5.31, -2.15) migraine days respect to VLCnKD (p < 0.0001). The 50% responder rate for migraine days was 74.28% (26/35 patients) during the VLCKD period, but only 8.57% (3/35 patients) during VLCnKD. Migraine attacks decreased by -3.02 (95% CI: -4.15, -1.88) during VLCKD respect to VLCnKD (p < 0.00001). There were no differences in the change of acute anti-migraine drug consumption (p = 0.112) and BMI (p = 0.354) between the 2 diets. A VLCKD has a preventive effect in overweight episodic migraine patients that appears within 1 month, suggesting that ketogenesis may be a useful therapeutic strategy for migraines.
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Grom

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Šefe

Vnosov: 63865

Pridružen: Če sep 23, 2004 18:35

Odgovor Sr sep 18, 2019 14:45

Re: Keto/Low carb študije

Ketogenic Diet and Skeletal Muscle Hypertrophy: a Frenemy Relationship?
Antonio Paoli


Abstract

Ketogenic diet (KD) is a nutritional regimen characterized by a high-fat and an adequate protein content and a very low carbohydrate level (less than 20 g per day or 5% of total daily energy intake). The insufficient level of carbohydrates forces the body to primarily use fat instead of sugar as a fuel source. Due to its characteristic, KD has often been used to treat metabolic disorders, obesity, cardiovascular disease, and type 2 diabetes. Skeletal muscle constitutes 40% of total body mass and is one of the major sites of glucose disposal. KD is a well-defined approach to induce weight loss, with its role in muscle adaptation and muscle hypertrophy less understood. Considering this lack of knowledge, the aim of this review was to examine the scientific evidence about the effects of KD on muscle hypertrophy. We first described the mechanisms of muscle hypertrophy per se, and secondly, we discussed the characteristics and the metabolic function of KD. Ultimately, we provided the potential mechanism that could explain the influence of KD on skeletal muscle hypertrophy.

Conclusions

Nutrients, growth factors, hormones, and muscle activity generate cellular signals that have the capacity to maintain and/or grow muscle mass. The regulation of skeletal muscle mass, in fact, reflects changes in protein synthesis and protein degradation, and several stimuli lead to protein synthesis and subsequent muscle hypertrophy. Indeed, nutrition is a key factor for muscle mass balance and macronutrients’ distribution, and the quantity may influence muscle growth and muscle response to training. The question that should be addressed is whether a diet in which one of the macronutrients is restricted, i.e. carbohydrates in KD, may affect skeletal muscle hypertrophy.

KD theoretically may affect skeletal muscle mass control pathways in several ways (Figure 2), but data provided by scientific literature suggest a negligible or no effect of KD on muscle mass with concomitant resistance training. KD may instead exert a protective effect against muscle mass loss during aging or during low calorie diets. The total effect seems to consist in the maintenance of muscle mass rather than a net hypertrophic effect.
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Grom

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Šefe

Vnosov: 63865

Pridružen: Če sep 23, 2004 18:35

Odgovor Ne nov 03, 2019 10:31

Re: Keto/Low carb študije

Če raziskave ugotavljajo, da je super za ameriško vojsko bo tudi za nas.. :)


Extended Ketogenic Diet and Physical Training Intervention in Military Personnel
Richard A LaFountain, Vincent J Miller, Emily C

Military Medicine, usz046, https://doi.org/10.1093/milmed/usz046

Abstract
Introduction

Ketogenic diets (KDs) that elevate ketones into a range referred to as nutritional ketosis represent a possible nutrition approach to address the emerging physical readiness and obesity challenge in the military. An emerging body of evidence demonstrates broad-spectrum health benefits attributed to being in nutritional ketosis, but no studies have specifically explored the use of a KD in a military population using daily ketone monitoring to personalize the diet prescription.

Materials and Methods
To evaluate the feasibility, metabolic, and performance responses of an extended duration KD, healthy adults (n = 29) from various military branches participated in a supervised 12-wk exercise training program. Fifteen participants self-selected to an ad libitum KD guided by daily measures of capillary blood ketones and 14 continued their normal mixed diet (MD). A battery of tests were performed before and after the intervention to assess changes in body mass, body composition, visceral fat, liver fat, insulin sensitivity, resting energy metabolism, and physical performance.
Results

All KD subjects were in nutritional ketosis during the intervention as assessed by daily capillary beta-hydroxybutyrate (βHB) (mean βHB 1.2 mM reported 97% of all days) and showed higher rates of fat oxidation indicative of keto-adaptation. Despite no instruction regarding caloric intake, the KD group lost 7.7 kg body mass (range −3.5 to −13.6 kg), 5.1% whole-body percent fat (range −0.5 to −9.6%), 43.7% visceral fat (range 3.0 to −66.3%) (all p < 0.001), and had a 48% improvement in insulin sensitivity; there were no changes in the MD group. Adaptations in aerobic capacity, maximal strength, power, and military-specific obstacle course were similar between groups (p > 0.05).

Conclusions
US military personnel demonstrated high adherence to a KD and showed remarkable weight loss and improvements in body composition, including loss of visceral fat, without compromising physical performance adaptations to exercise training. Implementation of a KD represents a credible strategy to enhance overall health and readiness of military service members who could benefit from weight loss and improved body composition.
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